Epithelial‐mesenchymal transition leads to crizotinib resistance in H2228 lung cancer cells with EML4‐ALK translocation - Kim - 2013 - Molecular Oncology - Wiley Online Library
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![PDF] Receptor ligand-triggered resistance to alectinib and its circumvention by Hsp90 inhibition in EML4-ALK lung cancer cells | Semantic Scholar](https://d3i71xaburhd42.cloudfront.net/2b265c0fd1b06a512472598fe7575a0e724931a3/2-Figure1-1.png "PDF] Receptor ligand-triggered resistance to alectinib and its circumvention by Hsp90 inhibition in EML4-ALK lung cancer cells | Semantic Scholar")
PDF] Receptor ligand-triggered resistance to alectinib and its circumvention by Hsp90 inhibition in EML4-ALK lung cancer cells | Semantic Scholar
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EMT changes and resistance to ALK-TKIs in H2228 cells exposed to... | Download Scientific Diagram
Differential protein stability and clinical responses ofEML4-ALK fusion variants to various ALK inhibitors in advancedALK-rearranged non-small cell lung cancer - Annals of Oncology
Sensitivities to ALK inhibitors (crizotinib and alectinib) in... | Download Scientific Diagram
EMT is associated with, but does not drive resistance to ALK inhibitors among EML4-ALK non-small cell lung cancer - ScienceDirect
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Epithelial‐mesenchymal transition leads to crizotinib resistance in H2228 lung cancer cells with EML4‐ALK translocation - Kim - 2013 - Molecular Oncology - Wiley Online Library
